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While some studies in PD models have revealed neuroprotective functions related to dopaminergic neuron survival, others have suggested a proinflammatory effect in LPS-treated models in which IL-4 exacerbates microglial activation and BBB disruption [187, 211]

Lack of inhibition of cell death by blocking caspases (e.g., Z-VAD-FMK)

The findings may suggest that these remedies may be used as potential therapeutic targets for HT treatment